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UGC NET Physical Education Subject -II Syllabus

The Subject -II syllabus for UGC NET Physical Education in full: 1 chapter, 13 topics and 16 sub-topics, every one listed below, plus 55 Subject -II flashcards with answers.

1Chapters
13Topics
16Sub-topics
55Flashcards

Subject -II syllabus: 1 chapter, 13 topics

Expand any chapter to see its topics and sub-topics.

  1. Exercise Physiology

    13 topics
    • Scope and Importance in the Field of Physical Education and Sports
    • Cardio-Respiratory Adaptations to Long and Short Term Physical Activities
    • Muscle
      • Types, Characteristics, and Functions
      • Microscopic Structure of Muscle Fibre
      • Sliding Filament Theory of Muscular Contraction
      • Types of Muscle Fibres and Sports Performance
      • Muscular Adaptations to Exercise
    • Neuro-Muscular Junction and Transmission of Nerve Impulse
    • Kinesthetic Sense Organs and Neural Control of Motor Skills
    • Biochemical Aspects of Exercise
      • Metabolism of Food Products
      • Aerobic and Anaerobic Systems during Rest and Exercise
      • Direct and Indirect Methods of Measuring Energy Cost of Exercise
    • Recovery Process
      • Physiological Aspects of Fatigue
      • Restoration of Energy Stores
      • Recovery Oxygen
      • Nutritional Aspects of Performance
    • Environmental Influence on Human Physiology under Exercise
    • Women in Sports
      • Trainability
      • Physiological Gender Differences and Special Problems of Women Athletes
    • Aging
      • Physiological Consequences
      • Lifestyle Management and Healthful Aging
    • Physiological Responses of Various Therapeutic Modalities and Rehabilitation
    • Physiological Aspects of Various Ergogenic Aids
    • Massage Manipulations and Their Physiological Responses

UGC NET Physical Education Subject -II flashcards — 55 questions with answers

  1. What is the scope and importance of exercise physiology in the field of Physical Education and Sports?

    Exercise physiology studies how the body's systems respond and adapt to acute and chronic physical activity. Its importance lies in designing scientific training programs, improving athletic performance, preventing injury, guiding rehabilitation, and promoting health and fitness.

  2. Define cardio-respiratory adaptation to physical activity.

    It is the structural and functional changes in the heart, blood vessels, and lungs that occur in response to physical activity, improving oxygen delivery to and carbon dioxide removal from working muscles.

  3. List the key short-term (acute) cardio-respiratory responses to exercise.

    Increased heart rate, increased stroke volume, increased cardiac output, rise in systolic blood pressure, redistribution of blood flow to muscles, increased breathing rate and tidal volume, and increased oxygen uptake.

  4. List the key long-term (chronic) cardio-respiratory adaptations to endurance training.

    Cardiac hypertrophy (larger left ventricle), increased stroke volume, lower resting heart rate (bradycardia), increased blood and plasma volume, increased capillary density, greater $\dot{V}O_{2max}$, and increased mitochondrial density.

  5. State the formula relating cardiac output to heart rate and stroke volume.

    $$Q = HR \times SV$$ where $Q$ is cardiac output, $HR$ is heart rate, and $SV$ is stroke volume.

  6. What is $\dot{V}O_{2max}$ and why is it important?

    $\dot{V}O_{2max}$ is the maximal rate of oxygen consumption during intense exercise. It is the single best indicator of aerobic (cardio-respiratory) endurance capacity.

  7. Give the Fick equation for oxygen consumption.

    $$\dot{V}O_{2} = Q \times (a\text{-}\bar{v})O_{2}\,\text{diff}$$ i.e. oxygen uptake equals cardiac output multiplied by the arterio-venous oxygen difference.

  8. What is the name given to the resting bradycardia of trained endurance athletes?

    Athlete's heart (sinus bradycardia), a lowered resting heart rate resulting from increased stroke volume and enhanced parasympathetic (vagal) tone.

  9. What are the three types of muscle in the human body?

    Skeletal (striated, voluntary), cardiac (striated, involuntary), and smooth (non-striated, involuntary) muscle.

  10. List the main characteristic properties of muscle tissue.

    Excitability (irritability), contractility, extensibility, and elasticity.

  11. State the main functions of skeletal muscle.

    Producing movement, maintaining posture and body position, stabilizing joints, and generating heat (thermogenesis).

  12. Describe the microscopic structure of a muscle fibre.

    A muscle fibre is a single multinucleated cell bounded by the sarcolemma, containing sarcoplasm, myofibrils made of actin and myosin, the sarcoplasmic reticulum, mitochondria, and an extensive T-tubule system.

  13. What is the sarcomere and what are its boundaries?

    The sarcomere is the functional contractile unit of a myofibril, extending from one Z-line (Z-disc) to the next Z-line.

  14. Identify the bands and zones of a sarcomere.

    The A-band (dark, contains myosin and overlapping actin), the I-band (light, actin only), the H-zone (myosin only, centre), the M-line (centre of H-zone), and the Z-line (boundary of sarcomere).

  15. Name the contractile proteins and the regulatory proteins of the muscle fibre.

    Contractile proteins: myosin (thick filament) and actin (thin filament). Regulatory proteins: tropomyosin and troponin.

  16. What is the sarcoplasmic reticulum's role in muscle contraction?

    It is a specialized endoplasmic reticulum that stores and releases calcium ions ($\ce{Ca^{2+}}$) which trigger contraction; its terminal cisternae release $\ce{Ca^{2+}}$ when stimulated.

  17. State the Sliding Filament Theory of muscular contraction.

    During contraction the thin (actin) filaments slide over the thick (myosin) filaments, drawing the Z-lines closer and shortening the sarcomere, while the actual filament lengths remain unchanged.

  18. Which structures shorten and which stay the same length during contraction per the sliding filament theory?

    The sarcomere, I-band, and H-zone shorten; the A-band and the lengths of the actin and myosin filaments themselves remain unchanged.

  19. Outline the cross-bridge cycle of muscle contraction.

    1) $\ce{Ca^{2+}}$ binds troponin, exposing actin sites; 2) myosin head binds actin forming a cross-bridge; 3) the power stroke pulls actin (ADP + $P_i$ released); 4) ATP binds myosin, detaching it; 5) ATP hydrolysis re-cocks the head for the next cycle.

  20. What role does $\ce{Ca^{2+}}$ play in initiating contraction?

    $\ce{Ca^{2+}}$ released from the sarcoplasmic reticulum binds to troponin, causing tropomyosin to move and expose the myosin-binding sites on actin, allowing cross-bridge formation.

Show the other 35 Subject -II flashcards
  1. What are the two principal classifications of muscle fibre types?

    Type I (slow-twitch, slow oxidative) and Type II (fast-twitch), with Type II subdivided into Type IIa (fast oxidative-glycolytic) and Type IIx/IIb (fast glycolytic).

  2. Compare slow-twitch (Type I) and fast-twitch (Type II) muscle fibres.

    Type I: slow contraction, high mitochondria/capillary density, fatigue-resistant, aerobic, red, suited to endurance. Type II: fast contraction, low oxidative capacity, fatigue quickly, anaerobic/glycolytic, white (IIx), suited to power and speed.

  3. Which fibre type predominates in elite marathon runners versus elite sprinters?

    Marathon (endurance) runners have a high proportion of Type I slow-twitch fibres; sprinters have a high proportion of Type II fast-twitch fibres.

  4. What muscular adaptations result from resistance (strength) training?

    Muscle fibre hypertrophy (increased cross-sectional area), increased myofibrillar protein and contractile proteins, increased anaerobic enzyme activity, and neural adaptations such as greater motor unit recruitment.

  5. What muscular adaptations result from endurance (aerobic) training?

    Increased mitochondrial number and size, increased oxidative enzyme activity, increased myoglobin content, increased capillary density, and enhanced fat utilization, mainly in Type I fibres.

  6. Distinguish muscle hypertrophy from hyperplasia.

    Hypertrophy is an increase in the size (cross-sectional area) of existing muscle fibres; hyperplasia is an increase in the number of fibres through fibre splitting (rare in humans).

  7. What is the neuromuscular junction?

    The neuromuscular junction is the synapse between the terminal of a motor neuron and the motor end-plate of a muscle fibre, where a nerve impulse is transmitted to the muscle.

  8. Which neurotransmitter is released at the neuromuscular junction and what is its action?

    Acetylcholine (ACh); it binds receptors on the motor end-plate, opening ion channels to depolarize the sarcolemma and generate an action potential in the muscle fibre.

  9. Describe the transmission of a nerve impulse across the neuromuscular junction.

    The nerve impulse triggers $\ce{Ca^{2+}}$ influx into the axon terminal, releasing acetylcholine into the synaptic cleft; ACh binds end-plate receptors causing depolarization, then acetylcholinesterase breaks down ACh to end the signal.

  10. What is a motor unit?

    A motor unit is a single motor neuron together with all the muscle fibres it innervates; it is the functional unit of muscle contraction.

  11. Define the 'all-or-none' law of muscle contraction.

    When stimulated at threshold or above, an individual muscle fibre (or motor unit) contracts maximally; below threshold it does not contract at all — there is no partial response.

  12. What are kinesthetic (proprioceptive) sense organs?

    Specialized sensory receptors that provide information about body position, movement, muscle tension, and joint angle, including muscle spindles, Golgi tendon organs, and joint receptors.

  13. Compare the functions of the muscle spindle and the Golgi tendon organ.

    The muscle spindle detects changes in muscle length and rate of stretch, triggering the stretch reflex; the Golgi tendon organ detects muscle tension and protects against excessive force by causing reflex relaxation (inverse stretch reflex).

  14. How does the nervous system exert neural control of motor skills?

    Through coordinated action of the motor cortex (planning and initiation), cerebellum (coordination, timing, balance), basal ganglia (movement regulation), spinal reflexes, and proprioceptive feedback for refined, skilled movement.

  15. What is meant by the biochemical aspects of exercise?

    The study of the chemical reactions and energy transformations — especially ATP production and breakdown of carbohydrates, fats, and proteins — that supply energy for muscular work.

  16. What is the immediate energy currency for muscular contraction?

    Adenosine triphosphate (ATP); its hydrolysis releases energy: $$\ce{ATP + H2O -> ADP + P_i + energy}$$

  17. Describe the metabolism of carbohydrates as an energy source.

    Carbohydrates are broken down to glucose, which undergoes glycolysis to pyruvate; aerobically pyruvate enters the Krebs cycle and electron transport chain, and anaerobically it is converted to lactic acid.

  18. Describe the metabolism of fats during exercise.

    Triglycerides are broken down (lipolysis) into glycerol and free fatty acids; fatty acids undergo beta-oxidation to acetyl-CoA, which enters the Krebs cycle, yielding large amounts of ATP aerobically.

  19. How are proteins used in energy metabolism?

    Proteins are broken into amino acids which can be deaminated and converted (via gluconeogenesis or directly) into intermediates for the Krebs cycle; they contribute minimally (about 5-10%) to energy, mainly during prolonged exercise.

  20. Name the three energy systems supplying ATP for muscular work.

    The ATP-PC (phosphagen) system, the anaerobic glycolytic (lactic acid) system, and the aerobic (oxidative) system.

  21. Describe the ATP-PC (phosphagen) energy system.

    An immediate anaerobic system in which phosphocreatine is broken down to rapidly resynthesize ATP: $$\ce{PC + ADP -> ATP + creatine}$$ Lasts about 8-10 seconds; fuels short, explosive efforts.

  22. Describe the anaerobic glycolytic (lactic acid) system.

    Glucose/glycogen is broken down without oxygen to produce ATP and lactic acid; it provides energy for high-intensity efforts lasting roughly 30 seconds to 2 minutes and yields a net 2 ATP per glucose.

  23. Describe the aerobic (oxidative) energy system.

    Using oxygen, carbohydrates and fats are fully oxidized via glycolysis, the Krebs cycle, and the electron transport chain, producing large amounts of ATP for prolonged, low-to-moderate intensity activity, with $\ce{CO2}$ and $\ce{H2O}$ as by-products.

  24. How much net ATP is produced from the complete aerobic oxidation of one glucose molecule?

    Approximately 36-38 ATP (commonly cited as 38) per molecule of glucose, compared with a net 2 ATP from anaerobic glycolysis.

  25. What is the energy systems' contribution at rest versus exercise?

    At rest the aerobic system predominates using mainly fat. During exercise contribution shifts toward carbohydrate and anaerobic systems as intensity rises; brief high-intensity work relies on ATP-PC and glycolysis.

  26. What is direct calorimetry for measuring energy cost of exercise?

    A method that measures energy expenditure directly by measuring the heat (in a sealed insulated chamber) produced by the body, since all metabolic energy ultimately appears as heat.

  27. What is indirect calorimetry for measuring energy cost of exercise?

    A method that estimates energy expenditure by measuring oxygen consumption and carbon dioxide production (gas exchange), since heat production is proportional to oxygen used (about 5 kcal per litre of $\ce{O2}$).

  28. Define the Respiratory Quotient (RQ) and give its formula.

    RQ is the ratio of carbon dioxide produced to oxygen consumed, indicating the fuel being used: $$RQ = \frac{\dot{V}CO_{2}}{\dot{V}O_{2}}$$ RQ of 1.0 indicates carbohydrate use, about 0.70 indicates fat use.

  29. Define the recovery process after exercise.

    Recovery is the set of physiological processes that restore the body to its pre-exercise (resting) state, including replenishing energy stores, removing metabolic by-products, and restoring oxygen and fluid balance.

  30. What are the physiological causes (aspects) of fatigue?

    Depletion of energy stores (PC and glycogen), accumulation of metabolites (lactic acid, $\ce{H+}$, $P_i$), disturbed ion balance, dehydration, central nervous system fatigue, and reduced neuromuscular transmission.

  31. Differentiate central fatigue from peripheral fatigue.

    Central fatigue originates in the central nervous system, reducing neural drive to the muscle; peripheral fatigue occurs within the muscle itself due to factors like substrate depletion and metabolite accumulation.

  32. Define EPOC (Excess Post-exercise Oxygen Consumption) or recovery oxygen.

    EPOC is the elevated oxygen consumption above resting level that continues after exercise, used to restore the body, traditionally described as the oxygen debt repaid during recovery.

  33. What are the two components of recovery oxygen (oxygen debt)?

    The fast (alactacid) component, which restores ATP-PC stores and reoxygenates myoglobin and blood; and the slow (lactacid) component, which removes/converts accumulated lactic acid.

  34. How are energy stores restored during recovery?

    Phosphocreatine is rapidly resynthesized (within a few minutes using aerobic energy), muscle and liver glycogen are replenished over hours through carbohydrate intake, and myoglobin/blood oxygen stores are restored quickly.

  35. What is the fate of lactic acid during recovery?

    Lactic acid is mostly oxidized to $\ce{CO2}$ and water for energy, converted back to glucose/glycogen in the liver via the Cori cycle (gluconeogenesis), or used by the heart and other tissues as fuel.

UGC NET Physical Education Subject -II FAQ

What is in the UGC NET Physical Education Subject -II syllabus?

UGC NET Physical Education Subject -II is split into 1 chapter: Exercise Physiology. Together they contain 13 topics and 16 sub-topics.

How many chapters are in UGC NET Physical Education Subject -II?

1 chapter, containing 13 topics and 16 sub-topics.

Are there flashcards for UGC NET Physical Education Subject -II?

Yes — a 55-card Subject -II deck, printed in full in the flashcards section of this page and available in the Examius app with spaced repetition. The free plan covers one exam of your choice, including its flashcards; Examius Pro, a one-time purchase, unlocks every other exam and deck.