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National Board Dental Examination / Integrated National Board Dental Examination (INBDE) Periodontics and Oral Surgery Syllabus

The Periodontics and Oral Surgery syllabus for National Board Dental Examination / Integrated National Board Dental Examination (INBDE) in full: 4 chapters, 15 topics and 14 sub-topics, every one listed below, plus 52 Periodontics and Oral Surgery flashcards with answers.

4Chapters
15Topics
14Sub-topics
52Flashcards

Periodontics and Oral Surgery syllabus: all 4 chapters

Expand any chapter to see its topics and sub-topics.

  1. Periodontal Anatomy and Disease

    4 topics
    • Healthy periodontium structure and function
    • Etiology and pathogenesis of periodontal disease
      • Microbial and host response factors
      • Systemic and behavioral risk factors
    • Classification of periodontal and peri-implant diseases
    • Gingival diseases and conditions
  2. Periodontal Therapy

    4 topics
    • Nonsurgical therapy and oral hygiene instruction
      • Scaling and root planing
      • Adjunctive antimicrobials
    • Periodontal surgery principles
      • Resective and regenerative procedures
      • Mucogingival and crown lengthening surgery
    • Periodontal maintenance and re-evaluation
    • Periodontal-systemic disease interrelationships
  3. Oral and Maxillofacial Surgery

    4 topics
    • Principles of exodontia and surgical extraction
      • Forceps technique and flap design
      • Impacted third molar management
    • Surgical complications and their management
      • Dry socket and hemorrhage
      • Nerve injury and oroantral communication
    • Odontogenic infections and their drainage
    • Biopsy techniques and lesion management
  4. Trauma, Implants, and Reconstruction

    3 topics
    • Dentoalveolar and maxillofacial trauma
      • Tooth fractures and avulsion management
      • Jaw fracture principles
    • Dental implant surgical placement and biology
      • Osseointegration concepts
      • Site assessment and grafting
    • Bone grafting and ridge preservation

National Board Dental Examination / Integrated National Board Dental Examination (INBDE) Periodontics and Oral Surgery flashcards — 52 questions with answers

  1. What are the four principal tissues that make up the periodontium?

    Gingiva, periodontal ligament (PDL), cementum, and alveolar bone.

  2. What is the normal biologic (supracrestal) width, and what are its two components?

    Approximately 2.04 mm total: ~0.97 mm junctional epithelium plus ~1.07 mm connective tissue attachment (Gargiulo). Violating it causes inflammation/bone loss.

  3. What are the principal fiber groups of the gingival connective tissue?

    Dentogingival, dentoperiosteal, alveologingival, circular, and transseptal fibers.

  4. Name the five principal fiber groups of the periodontal ligament.

    Alveolar crest, horizontal, oblique (most numerous, resist masticatory/axial forces), apical, and interradicular fibers.

  5. What type of epithelium is junctional epithelium, and how does it attach to the tooth?

    Nonkeratinized stratified squamous epithelium that attaches via hemidesmosomes and an internal basal lamina.

  6. What is the difference between cellular and acellular cementum and their locations?

    Acellular (primary) cementum covers the coronal/cervical root and anchors Sharpey's fibers; cellular (secondary) cementum covers the apical third/furcations and allows continuous deposition/repair.

  7. What is the keystone-pathogen hypothesis of periodontal disease, and which organism exemplifies it?

    Low-abundance keystone pathogens disrupt host-microbe homeostasis and remodel the biofilm into a dysbiotic state; Porphyromonas gingivalis is the prototypical keystone pathogen.

  8. Which three bacteria make up the 'red complex' associated with periodontitis?

    Porphyromonas gingivalis, Tannerella forsythia, and Treponema denticola (Socransky red complex).

  9. What is the primary etiologic agent of periodontal disease?

    Dysbiotic dental plaque biofilm; bacterial plaque is necessary, with host immune-inflammatory response mediating tissue destruction.

  10. What host-derived enzymes and cytokines drive periodontal tissue destruction?

    Matrix metalloproteinases (MMPs, especially collagenase/MMP-8), plus pro-inflammatory cytokines IL-1, IL-6, TNF-alpha, and PGE2 that promote bone resorption via the RANK/RANKL/OPG axis.

  11. Distinguish a periodontal pathogen of aggressive periodontitis in adolescents.

    Aggregatibacter actinomycetemcomitans (Aa), strongly associated with localized aggressive (molar/incisor) periodontitis; produces leukotoxin.

  12. In the 2017 AAP/EFP classification, how is periodontitis staged?

    Stages I-IV based on severity (interdental CAL, radiographic bone loss, tooth loss) and complexity (probing depth, furcation, ridge defects, need for rehabilitation).

  13. In the 2017 classification, how is periodontitis graded?

    Grades A, B, C based on rate of progression (bone loss/age ratio), with modifiers: smoking and diabetes (HbA1c).

  14. What three categories of periodontitis are recognized in the 2017 classification?

    Necrotizing periodontitis, periodontitis as a manifestation of systemic disease, and periodontitis (the single category replacing chronic and aggressive).

  15. What are the two clinical health categories for the periodontium in the 2017 scheme?

    Clinical health on an intact periodontium and clinical health on a reduced periodontium (in a stable periodontitis patient or non-periodontitis patient).

  16. What defines peri-implant mucositis versus peri-implantitis?

    Peri-implant mucositis: inflammation (bleeding on probing) without progressive bone loss; peri-implantitis: inflammation PLUS progressive marginal bone loss beyond initial remodeling.

  17. What are the classic clinical features of plaque-induced gingivitis?

    Erythema, edema, bleeding on probing, no clinical attachment loss or bone loss; fully reversible with plaque control.

  18. What characterizes Necrotizing Ulcerative Gingivitis (NUG)?

    Punched-out, necrotic ('cratered') interdental papillae, pain, spontaneous bleeding, pseudomembrane, and fetor oris; associated with stress, smoking, immunosuppression, and fusospirochetal flora.

  19. What systemic condition should be suspected with necrotizing periodontal disease (NUG/NUP)?

    Immunosuppression, notably HIV/AIDS (CD4 count); also severe malnutrition (noma in children).

  20. Which drugs classically cause gingival enlargement (overgrowth)?

    Phenytoin (anticonvulsant), cyclosporine (immunosuppressant), and calcium channel blockers (e.g., nifedipine).

Show the other 32 Periodontics and Oral Surgery flashcards
  1. What is the primary goal and main instrument category of nonsurgical periodontal therapy?

    Disruption/removal of subgingival biofilm and calculus to reduce inflammation; performed by scaling and root planing (SRP) with hand curettes (e.g., Gracey) and ultrasonic scalers.

  2. What is the difference between scaling and root planing?

    Scaling removes plaque and calculus from crown/root surfaces; root planing additionally removes contaminated cementum/dentin to leave a smooth, biocompatible root surface.

  3. Which area-specific Gracey curettes are used for posterior buccal/lingual versus mesial versus distal surfaces?

    Gracey 7/8 (and 11/12) for buccal/lingual; Gracey 11/12 for mesial surfaces; Gracey 13/14 for distal surfaces of posterior teeth.

  4. What is the recommended toothbrushing technique for sulcular plaque removal?

    The modified Bass technique: bristles angled 45 degrees to the long axis into the gingival sulcus with short vibratory/circular strokes.

  5. What is the rationale and timing for periodontal re-evaluation after SRP?

    Re-evaluate ~4-6 weeks after SRP to allow tissue healing, then reassess probing depths, BOP, and attachment to decide on maintenance versus surgical therapy.

  6. What is the main objective of periodontal flap surgery?

    To gain access for root debridement and to reduce/eliminate pockets while preserving or regenerating attachment, allowing visualization of root surfaces and osseous defects.

  7. Differentiate gingivectomy from apically positioned flap.

    Gingivectomy excises and removes pocket wall (used for suprabony pockets/gingival overgrowth with adequate keratinized tissue); apically positioned flap repositions tissue apically to reduce pockets while preserving keratinized gingiva.

  8. What is guided tissue regeneration (GTR) and its principle?

    Use of a barrier membrane to exclude epithelium/gingival connective tissue, allowing PDL and bone cells to repopulate the defect and regenerate true attachment; works best in narrow 3-wall intrabony defects and Class II furcations.

  9. What biologic agent derived from porcine tooth buds is used to promote periodontal regeneration?

    Enamel matrix derivative (EMD, e.g., Emdogain), which mimics developmental cementogenesis to stimulate periodontal regeneration.

  10. How are furcation involvements classified (Glickman)?

    Grade I: incipient/horizontal loss within enamel; Grade II: cul-de-sac, partial horizontal loss; Grade III: through-and-through but soft-tissue covered; Grade IV: through-and-through and clinically visible.

  11. What is the recommended periodontal maintenance recall interval for most treated periodontitis patients?

    Every 3 months (3-month recall) for supportive periodontal therapy, adjusted by risk and disease control.

  12. What does supportive periodontal therapy (maintenance) typically include?

    Update of medical/dental history, plaque/BOP assessment, full periodontal charting, oral hygiene reinforcement, supra/subgingival debridement, and determination of recall interval.

  13. How does diabetes mellitus interrelate with periodontal disease (bidirectional relationship)?

    Diabetes (especially poor glycemic control) increases periodontitis severity via AGEs and exaggerated inflammation; conversely, periodontitis worsens glycemic control. Periodontal treatment can modestly lower HbA1c.

  14. What is the proposed link between periodontitis and cardiovascular disease?

    Chronic periodontal infection contributes to systemic inflammation, bacteremia, and atherogenesis; periodontal pathogens have been found in atheromatous plaques (association, not proven causation).

  15. What adverse pregnancy outcome is associated with maternal periodontitis?

    Increased risk of preterm low birth weight, thought to be mediated by inflammatory mediators (PGE2, IL-1) and bacteremia.

  16. What is the recommended preoperative chair/patient positioning consideration for maxillary versus mandibular extractions?

    For maxillary extractions the occlusal plane is ~60 degrees to the floor with the chair higher; for mandibular extractions the occlusal plane is parallel to the floor with the chair lower (near operator elbow level).

  17. What two basic mechanical principles govern simple (forceps) exodontia?

    Use of the tooth as a lever and the expansion of the bony socket; controlled forces (apical, buccal-lingual, rotational, tractional) luxate and deliver the tooth.

  18. What is the function of a dental elevator, and what three working principles does it use?

    To luxate teeth/roots and expand bone; it uses the lever (most common), wheel-and-axle, and wedge principles.

  19. When is a surgical (open) extraction with flap and bone removal indicated?

    For impacted/ankylosed teeth, hypercementosis, divergent or curved roots, root fracture during simple extraction, thick/dense bone, or thin fragile roots likely to fracture.

  20. What are the cardinal signs and management of a dry socket (alveolar osteitis)?

    Severe throbbing pain 2-4 days post-extraction with empty/denuded socket and fetid odor due to lost clot. Management: gentle irrigation and medicated (eugenol) dressing for palliation; not an infection, so antibiotics are not routinely needed.

  21. How is an oroantral communication managed based on size?

    <2 mm: clot stabilization, no surgical closure; 2-6 mm: figure-of-eight suture and sinus precautions, antibiotics, decongestants; >6 mm: surgical flap closure (buccal advancement or buccal fat pad).

  22. What is the immediate management if a tooth or root tip is displaced into the maxillary sinus?

    Attempt retrieval via the socket with suction/irrigation; if unsuccessful, refer for surgical retrieval (e.g., Caldwell-Luc or endoscopic approach) and provide antibiotics/decongestants/sinus precautions.

  23. What is the fundamental surgical principle for managing an odontogenic infection with an abscess?

    Incision and drainage (I&D) to establish drainage, plus removal of the source (extraction or endodontic treatment); antibiotics are adjunctive, not a substitute for drainage.

  24. What is Ludwig's angina and why is it an emergency?

    A rapidly spreading bilateral cellulitis of the submandibular, sublingual, and submental spaces; it threatens the airway by elevating/displacing the tongue, requiring airway management, IV antibiotics, and surgical drainage.

  25. Which fascial space is involved when a mandibular molar infection perforates lingual to the mylohyoid muscle versus buccal/below it?

    Above the mylohyoid attachment (or roots above it) drains to the sublingual space; below the mylohyoid attachment drains to the submandibular space (second/third molar roots typically below).

  26. Differentiate excisional from incisional biopsy and when each is indicated.

    Excisional removes the entire lesion with a margin (small <1 cm, benign-appearing lesions); incisional removes a representative portion (large lesions or suspected malignancy) to establish diagnosis before definitive treatment.

  27. What is the appropriate management/biopsy approach for a suspected malignant oral lesion that does not resolve in 2 weeks?

    Incisional biopsy including a margin of normal tissue (avoid necrotic center), or referral; persistent ulcer/leukoplakia >2 weeks warrants biopsy to rule out squamous cell carcinoma.

  28. What is the recommended management sequence (ABCDE) and priority for a patient with maxillofacial trauma?

    Follow ATLS: Airway (with C-spine control), Breathing, Circulation/hemorrhage control, Disability, Exposure — life-threatening issues before definitive fracture repair.

  29. What is the recommended storage medium and time limit for an avulsed permanent tooth before replantation?

    Replant immediately if possible; otherwise store in Hank's Balanced Salt Solution, cold milk, or saliva. PDL cells begin to die after ~30-60 minutes of dry time; avoid water and scrubbing the root.

  30. What defines successful osseointegration of a dental implant (Branemark concept)?

    Direct structural and functional connection between living bone and the implant surface without intervening fibrous tissue, clinically immobile and asymptomatic under load.

  31. What is the difference between primary (mechanical) and secondary (biological) implant stability?

    Primary stability is the mechanical engagement of the implant with bone at placement (no micromotion); secondary stability develops from osseointegration (new bone formation) over healing, replacing primary stability.

  32. Compare autograft, allograft, xenograft, and alloplast in bone grafting.

    Autograft (patient's own bone): osteogenic, osteoinductive, osteoconductive (gold standard); allograft (human donor, e.g., FDBA/DFDBA): osteoinductive/conductive; xenograft (e.g., bovine, Bio-Oss): osteoconductive scaffold; alloplast (synthetic, e.g., HA/TCP): osteoconductive only.

National Board Dental Examination / Integrated National Board Dental Examination (INBDE) Periodontics and Oral Surgery FAQ

What is in the National Board Dental Examination / Integrated National Board Dental Examination (INBDE) Periodontics and Oral Surgery syllabus?

National Board Dental Examination / Integrated National Board Dental Examination (INBDE) Periodontics and Oral Surgery is split into 4 chapters: Periodontal Anatomy and Disease, Periodontal Therapy, Oral and Maxillofacial Surgery and Trauma, Implants, and Reconstruction. Together they contain 15 topics and 14 sub-topics.

How many chapters are in National Board Dental Examination / Integrated National Board Dental Examination (INBDE) Periodontics and Oral Surgery?

4 chapters, containing 15 topics and 14 sub-topics. The largest chapter is Periodontal Anatomy and Disease, with 4 topics.

Are there flashcards for National Board Dental Examination / Integrated National Board Dental Examination (INBDE) Periodontics and Oral Surgery?

Yes — a 52-card Periodontics and Oral Surgery deck, printed in full in the flashcards section of this page and available in the Examius app with spaced repetition. The free plan covers one exam of your choice, including its flashcards; Examius Pro, a one-time purchase, unlocks every other exam and deck.