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Fellowship of the Royal College of Surgeons (FRCS) Trauma and Orthopaedic Surgery Syllabus

The Trauma and Orthopaedic Surgery syllabus for Fellowship of the Royal College of Surgeons (FRCS) in full: 4 chapters, 19 topics and 25 sub-topics, every one listed below, plus 51 Trauma and Orthopaedic Surgery flashcards with answers.

4Chapters
19Topics
25Sub-topics
51Flashcards

Trauma and Orthopaedic Surgery syllabus: all 4 chapters

Expand any chapter to see its topics and sub-topics.

  1. Principles of Fracture Management

    5 topics
    • Fracture classification and healing
      • Patterns of fracture and biomechanics
      • Primary versus secondary bone healing
      • Delayed union and non-union
    • Principles of fracture fixation
      • Conservative management and casting
      • Internal fixation and AO principles
      • External fixation
    • Open fractures and soft tissue management
      • Gustilo-Anderson classification
      • Antibiotic and debridement principles
    • Compartment syndrome
    • Polytrauma and damage control orthopaedics
  2. Adult Elective Orthopaedics

    5 topics
    • Osteoarthritis and joint replacement
      • Hip arthroplasty principles and complications
      • Knee arthroplasty
      • Periprosthetic infection and loosening
    • Inflammatory arthritis surgical management
    • Sports injuries of the knee
      • Ligament and meniscal injuries
    • Shoulder and elbow disorders
    • Foot and ankle pathology
  3. Spinal Disorders

    4 topics
    • Degenerative spinal disease
      • Disc prolapse and radiculopathy
      • Spinal stenosis
    • Spinal trauma and cord injury
      • Spinal stability assessment
      • Cauda equina syndrome
    • Spinal infection and tumours
    • Spinal deformity
  4. Specialist Orthopaedics

    5 topics
    • Paediatric orthopaedics
      • Developmental dysplasia of the hip
      • Perthes and slipped upper femoral epiphysis
      • Paediatric fractures and the growth plate
    • Hand and wrist surgery
      • Carpal tunnel and nerve compression
      • Hand trauma and tendon injuries
    • Bone and soft tissue tumours
      • Benign and malignant bone tumours
      • Metastatic bone disease
    • Orthopaedic infection
      • Osteomyelitis
      • Septic arthritis
    • Peripheral nerve injury and repair

Fellowship of the Royal College of Surgeons (FRCS) Trauma and Orthopaedic Surgery flashcards — 51 questions with answers

  1. What four factors form the basis of the Gustilo-Anderson classification of open fractures?

    Wound size, degree of soft tissue damage, level of contamination, and the presence of vascular injury. Type I: wound <1 cm, clean. Type II: 1-10 cm, moderate soft tissue damage. Type IIIA: extensive soft tissue but adequate coverage. Type IIIB: extensive soft tissue loss requiring flap coverage. Type IIIC: any open fracture with arterial injury requiring repair.

  2. Describe the stages of secondary (endochondral) fracture healing in order.

    1) Haematoma formation and inflammation (haematoma, cytokine release). 2) Soft callus formation (fibrocartilage, days to ~2-3 weeks). 3) Hard callus formation (woven bone via endochondral and intramembranous ossification). 4) Remodelling (woven bone replaced by lamellar bone along stress lines via Wolff's law). Occurs with relative stability.

  3. What is the difference between primary and secondary bone healing?

    Primary (direct) healing occurs with absolute stability (rigid fixation, <2% strain) and no callus, via cutting cones / Haversian remodelling across the fracture gap (contact or gap healing). Secondary (indirect) healing occurs with relative stability (some interfragmentary motion) and proceeds through callus formation by endochondral ossification.

  4. State Perren's strain theory and the strain thresholds for bone healing.

    Interfragmentary strain = change in gap length divided by original gap length: $\varepsilon = \frac{\Delta L}{L}$. Granulation tissue tolerates up to ~100% strain, cartilage ~10%, and lamellar bone <2%. Bone cannot form if local strain exceeds ~2%; this governs whether absolute or relative stability is needed.

  5. What are the AO principles of fracture fixation?

    1) Anatomical reduction (especially intra-articular fractures). 2) Stable fixation appropriate to the fracture (absolute or relative stability). 3) Preservation of blood supply to bone and soft tissues. 4) Early, active, pain-free mobilisation of the patient and limb (function).

  6. How does a lag screw produce absolute stability, and what is the principle of overdrilling?

    A lag screw compresses two fragments together. The near cortex is overdrilled to the screw's outer (thread) diameter (gliding hole) so threads do not engage it, while the far cortex is drilled to core diameter (threaded hole). Tightening pulls the far fragment toward the near cortex, generating interfragmentary compression and absolute stability.

  7. Compare absolute and relative stability in terms of construct, strain, and healing.

    Absolute stability: interfragmentary compression (lag screw, compression plate), near-zero motion (<2% strain), heals by primary (direct) bone healing with no callus. Relative stability: controlled micromotion (bridging plate, intramedullary nail, external fixator), heals by secondary healing with callus.

  8. What is compartment syndrome and what is the critical perfusion concept behind it?

    A condition where raised pressure within a closed osseofascial compartment compromises tissue perfusion, leading to ischaemia. Perfusion depends on the difference between diastolic blood pressure and compartment pressure; a low perfusion pressure (delta pressure) causes capillary collapse and tissue necrosis even with palpable distal pulses.

  9. What delta pressure threshold indicates fasciotomy in compartment syndrome, and what is the clinical hallmark?

    Fasciotomy is indicated when delta pressure (diastolic BP minus compartment pressure) is $\leq 30$ mmHg. The earliest and most reliable clinical sign is pain out of proportion, worsened by passive stretch of the muscles in the compartment. Pulselessness and paralysis are late signs.

  10. List the classic clinical features (the P's) of compartment syndrome and which are early vs late.

    Pain (out of proportion, on passive stretch) - early and most reliable; Paraesthesia - early-intermediate; Pallor, Pulselessness, Paralysis, Poikilothermia (cold) - late and unreliable signs indicating established ischaemia. Diagnosis should be made before late signs appear.

  11. What is damage control orthopaedics (DCO) and when is it preferred over early total care?

    DCO is temporary stabilisation (usually external fixation), control of haemorrhage and contamination, with definitive fixation delayed until the patient is physiologically stable. It is preferred in unstable/borderline polytrauma patients to avoid a 'second hit' that worsens the systemic inflammatory response (SIRS). Early total care suits stable patients.

  12. What physiological parameters define an unstable polytrauma patient suitable for damage control?

    Markers of the 'lethal triad' and shock: persistent hypotension, lactate >2.5 mmol/L, base deficit, hypothermia (<35 C), coagulopathy, low platelets, and severe chest/head/abdominal injury. These patients are at high risk of the second hit; definitive long-bone surgery is deferred.

  13. Define osteoarthritis and list its four cardinal radiographic features.

    A degenerative, non-inflammatory disorder of synovial joints with focal loss of articular cartilage and reactive bone change. Radiographic features (LOSS): Loss of joint space (asymmetrical), Osteophytes, Subchondral Sclerosis, Subchondral cysts (geodes).

  14. Contrast the radiographic features of osteoarthritis and rheumatoid arthritis.

    OA: asymmetrical joint space loss, osteophytes, subchondral sclerosis and cysts, no osteopenia. RA: symmetrical (concentric) joint space loss, periarticular osteopenia, marginal erosions, soft tissue swelling, and absence of osteophytes/sclerosis. RA is inflammatory and erosive; OA is degenerative and productive.

  15. What are the main bearing surface options in total hip replacement and a key drawback of each?

    Metal-on-polyethylene (cheap, reliable; polyethylene wear and osteolysis). Ceramic-on-polyethylene (low wear; cost). Ceramic-on-ceramic (lowest wear, hard; risk of squeaking and fracture of ceramic). Metal-on-metal (largely abandoned due to metal ion release / ALVAL / pseudotumours).

  16. What is the main surgical principle of joint replacement for inflammatory arthritis, and a key perioperative concern?

    Surgery (synovectomy, arthroplasty, arthrodesis, tendon repair) aims to relieve pain, restore function and prevent deformity once medical control fails. Key concerns: cervical spine instability (atlantoaxial subluxation) risking cord injury at intubation, increased infection risk from immunosuppression/DMARDs, and poor bone/soft tissue quality.

  17. Which knee structure is the primary restraint to anterior tibial translation, and what test assesses it?

    The anterior cruciate ligament (ACL) is the primary restraint to anterior translation of the tibia on the femur. It is assessed by the Lachman test (most sensitive), the anterior drawer test, and the pivot-shift test (assesses rotational/functional instability).

  18. Describe the 'unhappy triad' of the knee and its mechanism.

    Classically a valgus force with rotation (e.g., lateral blow to a planted leg) injuring the ACL, the medial collateral ligament (MCL), and a meniscus. Originally described as medial meniscus, but the lateral meniscus is now recognised as more commonly involved with this mechanism.

  19. What is the difference between primary and secondary impingement of the shoulder?

    Primary (subacromial) impingement is structural narrowing of the subacromial space (e.g., type III hooked acromion, osteophytes) causing rotator cuff compression. Secondary impingement results from glenohumeral instability or scapular dyskinesia causing dynamic narrowing, typically in younger overhead athletes.

  20. What nerve is most commonly injured in anterior shoulder dislocation, and how is it tested?

    The axillary nerve (C5-C6), injured as it passes around the surgical neck of the humerus through the quadrangular space. Tested by sensation over the 'regimental badge' area (lateral upper arm over deltoid) and deltoid motor function (abduction). Always assess before and after reduction.

Show the other 31 Trauma and Orthopaedic Surgery flashcards
  1. What is the 'terrible triad' of the elbow?

    Posterolateral rotatory elbow dislocation with: 1) radial head fracture, 2) coronoid process fracture, and 3) lateral collateral ligament (LCL) complex injury. It is highly unstable and usually requires surgical reconstruction to restore the bony and ligamentous stabilisers.

  2. What is hallux valgus and what radiographic angles define it?

    Lateral deviation of the great toe at the first MTP joint with medial deviation of the first metatarsal. Defined by the hallux valgus angle (HVA, normal $<15^{\circ}$) and the intermetatarsal angle (IMA, normal $<9^{\circ}$). Surgical procedure choice (e.g., scarf/chevron vs basal osteotomy/Lapidus) depends on the IMA severity.

  3. Describe the Lauge-Hansen classification concept for ankle fractures.

    It classifies ankle fractures by the foot position and the direction of the deforming force, in sequential stages. The two-word name gives foot position then force direction: e.g., Supination-External Rotation (most common), Supination-Adduction, Pronation-External Rotation, Pronation-Abduction. It predicts the pattern of bony and ligamentous injury.

  4. What characterises the pain of lumbar spinal stenosis (neurogenic claudication) and how is it distinguished from vascular claudication?

    Neurogenic claudication: leg pain/heaviness on walking and standing, relieved by lumbar flexion (sitting, leaning forward, walking uphill), with preserved pulses. Vascular claudication: relieved by standing still (rest alone), worse uphill, with absent pulses. Stenosis patients can cycle/push a trolley comfortably (flexed posture).

  5. In lumbar disc herniation, which nerve root is typically compressed and why?

    A paracentral (posterolateral) disc herniation compresses the traversing (lower) nerve root, e.g., an L4/5 disc compresses the L5 root. A far-lateral (foraminal) herniation compresses the exiting root at that level (L4 root at L4/5). The traversing root is most commonly affected.

  6. What is cauda equina syndrome and what are its red-flag features requiring urgent MRI and decompression?

    Compression of the lumbosacral nerve roots below the conus. Red flags: bilateral sciatica, saddle (perineal) anaesthesia, bladder/bowel dysfunction (urinary retention with overflow incontinence), reduced anal tone, and sexual dysfunction. It is a surgical emergency; urgent MRI and decompression (ideally <48 h) are required.

  7. How does the SLIC classification guide management of subaxial cervical spine trauma?

    The Subaxial Injury Classification (SLIC) scores three components: injury morphology, disco-ligamentous complex (DLC) integrity, and neurological status. Total score $\leq 3$ suggests non-operative management, $=4$ is indeterminate, and $\geq 5$ suggests surgical stabilisation.

  8. Distinguish complete from incomplete spinal cord injury and the role of spinal shock.

    Complete injury: no motor or sensory function below the level, including no sacral sparing (S4-5). Incomplete: some preserved function below the level (e.g., sacral sparing - perianal sensation, voluntary anal contraction). The completeness can only be confirmed after spinal shock resolves, marked by return of the bulbocavernosus reflex.

  9. Describe central cord syndrome: mechanism, deficit pattern, and prognosis.

    Most common incomplete cord syndrome, typically from a hyperextension injury in an older patient with pre-existing cervical spondylosis. Causes greater weakness in the upper limbs than the lower limbs (centrally located cervical motor tracts), with variable sensory loss and bladder dysfunction. Prognosis for recovery is generally good but often incomplete.

  10. Contrast anterior cord syndrome and Brown-Sequard syndrome.

    Anterior cord syndrome: anterior spinal artery territory; loss of motor and pain/temperature below the lesion with preserved proprioception/vibration (dorsal columns spared); poor prognosis. Brown-Sequard (cord hemisection): ipsilateral loss of motor and proprioception/vibration, contralateral loss of pain/temperature; best prognosis of the incomplete syndromes.

  11. What organism most commonly causes pyogenic vertebral osteomyelitis, and what investigation is most sensitive?

    Staphylococcus aureus is the most common organism. MRI with contrast is the most sensitive and specific imaging investigation (showing disc and endplate enhancement, paravertebral/epidural collections). Blood cultures and image-guided biopsy identify the organism to guide targeted antibiotic therapy.

  12. Which primary cancers most commonly metastasise to the spine, and which are typically lytic vs sclerotic?

    Breast, prostate, lung, kidney (renal), and thyroid. Predominantly lytic: lung, kidney, thyroid. Sclerotic (blastic): prostate (and breast can be mixed). Mnemonic 'BLT with Kosher Pickle': Breast, Lung, Thyroid, Kidney, Prostate. The spine is the most common site of skeletal metastasis.

  13. How is a scoliosis curve measured and what magnitude generally triggers surgical consideration in adolescent idiopathic scoliosis?

    By the Cobb angle, measured between lines drawn along the end-plates of the most tilted upper and lower vertebrae of the curve. A curve $>10^{\circ}$ defines scoliosis; bracing is considered for curves ~$25\text{-}40^{\circ}$ in skeletally immature patients, and surgery (fusion) is generally considered for curves $>45\text{-}50^{\circ}$.

  14. What is Risser sign and why is it important in scoliosis management?

    The Risser sign grades skeletal maturity by the ossification of the iliac apophysis from lateral to medial, graded 0 (no ossification) to 5 (complete fusion to iliac crest). Lower grades indicate greater remaining growth and higher risk of curve progression, guiding the timing of bracing versus surgery.

  15. What is developmental dysplasia of the hip (DDH) and which clinical tests screen for it in neonates?

    Abnormal development of the hip with instability/subluxation/dislocation and a shallow acetabulum. Screened with the Ortolani test (reduces a dislocated hip - 'O' for out-to-in/relocate with a clunk) and the Barlow test (dislocates a reducible hip). Ultrasound is the imaging modality of choice under ~4-6 months.

  16. What is slipped capital femoral epiphysis (SCFE), its typical patient, and management principle?

    Displacement of the femoral epiphysis (head) posteriorly and inferiorly relative to the metaphysis through the physis, typically in an overweight adolescent (10-16 years), presenting with hip/knee pain and obligatory external rotation on flexion. Treatment is urgent in-situ pinning to prevent progression and avascular necrosis; the slip is unstable if weight-bearing is not possible.

  17. Describe the typical course of Legg-Calve-Perthes disease and a key prognostic factor.

    Idiopathic avascular necrosis of the femoral head in children (typically 4-8 years), progressing through necrosis, fragmentation, reossification, and remodelling. Key prognostic factors: age at onset (younger = better), and degree of femoral head involvement/congruency (lateral pillar/Herring classification). Aim is to keep the femoral head contained and spherical.

  18. What is carpal tunnel syndrome, the nerve and motor sign involved, and provocative tests?

    Compression of the median nerve at the wrist beneath the flexor retinaculum. Causes paraesthesia in the radial 3.5 digits and thenar wasting (abductor pollicis brevis weakness). Provocative tests: Tinel's sign (tapping over the nerve) and Phalen's test (sustained wrist flexion). Nerve conduction studies confirm the diagnosis.

  19. What is Dupuytren's contracture and which structures/joints are typically involved?

    A progressive fibroproliferative disorder of the palmar fascia with myofibroblast proliferation forming nodules and cords, causing flexion contracture, most commonly of the ring and little fingers at the MCP and PIP joints. Associated with northern European descent, alcohol, diabetes, and a positive family history (Garrod's pads, Ledderhose, Peyronie's).

  20. Why is a scaphoid fracture prone to avascular necrosis and non-union?

    The scaphoid receives its blood supply predominantly retrograde from branches of the radial artery entering distally (dorsal ridge). Therefore proximal pole fractures interrupt the blood supply to the proximal fragment, leading to a high risk of avascular necrosis and non-union, especially with delayed treatment.

  21. How does the Enneking (surgical staging) system classify musculoskeletal tumours?

    Benign lesions are staged 1 (latent), 2 (active), 3 (aggressive). Malignant lesions are staged by grade (low = I, high = II) and compartment status (A = intracompartmental, B = extracompartmental), with stage III denoting metastatic disease. It guides the required surgical margin (intralesional, marginal, wide, radical).

  22. List the surgical margins for tumour resection and where each plane lies.

    Intralesional (through the tumour - debulking), Marginal (through the reactive zone/pseudocapsule - risk of residual satellite cells), Wide (through normal tissue beyond the reactive zone, leaving a cuff of normal tissue), and Radical (removal of the entire compartment containing the tumour). Malignant tumours require at least a wide margin.

  23. Name the most common primary bone sarcomas and their typical age and location.

    Osteosarcoma: adolescents/young adults, metaphysis of long bones (distal femur, proximal tibia), 'sunburst' periosteal reaction and Codman's triangle. Ewing's sarcoma: children/adolescents, diaphysis, 'onion-skin' periosteal reaction, t(11;22) translocation. Chondrosarcoma: older adults, axial skeleton/pelvis, cartilage-forming.

  24. What red-flag features in a soft tissue lump suggest sarcoma and mandate referral?

    A lump that is larger than 5 cm, deep to fascia, rapidly growing, painful, or recurrent after previous excision. Any such lump warrants urgent referral to a specialist sarcoma centre for MRI and biopsy (planned through the future resection field) before any excision - avoid 'whoops' unplanned excisions.

  25. Define osteomyelitis and contrast the typical routes/patterns in children vs adults.

    Infection of bone and marrow. In children it is usually haematogenous, affecting the metaphysis of long bones (slow blood flow in sinusoidal loops). In adults it is more often contiguous (open fracture, surgery, diabetic foot) or secondary to vascular insufficiency. Staph aureus is the most common organism overall.

  26. What is the gold-standard treatment principle for chronic osteomyelitis, and what is an involucrum vs sequestrum?

    Thorough surgical debridement of all dead/infected tissue (excision of sequestrum), dead-space management, stable skeleton, soft tissue coverage, plus targeted antibiotics. A sequestrum is a fragment of dead, avascular bone; an involucrum is the new periosteal bone formed around it. Antibiotics alone cannot eradicate infection harboured in avascular sequestra.

  27. How is native (non-prosthetic) septic arthritis diagnosed and managed, and which classic criteria aid the knee?

    Diagnosis by urgent joint aspiration: turbid fluid, raised WCC (commonly >50,000/mm3 with neutrophil predominance), Gram stain and culture. Kocher criteria (paediatric hip) use non-weight-bearing, fever >38.5C, ESR >40, WCC >12,000. Management: urgent surgical washout/arthroscopic lavage and IV antibiotics to prevent rapid cartilage destruction.

  28. Describe the Seddon classification of peripheral nerve injury.

    Neurapraxia: local conduction block, axon intact, full recovery (myelin injury). Axonotmesis: axon disrupted with Wallerian degeneration but endoneurial tubes/connective tissue intact, recovery by regeneration at ~1 mm/day. Neurotmesis: complete transection of the nerve including connective tissue, no spontaneous recovery, requires surgical repair.

  29. What is the rate of peripheral nerve regeneration, and what principle guides timing of nerve repair?

    Axonal regeneration proceeds at approximately 1 mm/day (about 1 inch/month). Sharp, clean transections are best repaired primarily (within 72 hours). Tidy/blunt injuries are repaired early; contaminated or crush injuries are tagged and repaired at ~3 weeks once the zone of injury declares itself, before motor end-plates degenerate (~12-18 months).

  30. What is a Tscherne classification used for, and why does it matter in closed fractures?

    The Tscherne classification grades the severity of the soft tissue injury in closed fractures (C0 minimal to C3 severe with compartment syndrome/vascular injury). It matters because the soft tissue envelope, not just the bony injury, dictates timing of surgery - severe swelling/blistering may mandate delay or temporary spanning external fixation before definitive fixation.

  31. State Wolff's law and Hueter-Volkmann law and their orthopaedic relevance.

    Wolff's law: bone remodels and adapts its architecture in response to the mechanical loads placed upon it (load increases density along stress lines; disuse causes resorption). Hueter-Volkmann law: increased compression across a physis inhibits growth and reduced/tension stimulates it - explaining progression of angular deformity and the rationale for guided growth (hemiepiphysiodesis).

Fellowship of the Royal College of Surgeons (FRCS) Trauma and Orthopaedic Surgery FAQ

What is in the Fellowship of the Royal College of Surgeons (FRCS) Trauma and Orthopaedic Surgery syllabus?

Fellowship of the Royal College of Surgeons (FRCS) Trauma and Orthopaedic Surgery is split into 4 chapters: Principles of Fracture Management, Adult Elective Orthopaedics, Spinal Disorders and Specialist Orthopaedics. Together they contain 19 topics and 25 sub-topics.

How many chapters are in Fellowship of the Royal College of Surgeons (FRCS) Trauma and Orthopaedic Surgery?

4 chapters, containing 19 topics and 25 sub-topics. The largest chapter is Principles of Fracture Management, with 5 topics.

Are there flashcards for Fellowship of the Royal College of Surgeons (FRCS) Trauma and Orthopaedic Surgery?

Yes — a 51-card Trauma and Orthopaedic Surgery deck, printed in full in the flashcards section of this page and available in the Examius app with spaced repetition. The free plan covers one exam of your choice, including its flashcards; Examius Pro, a one-time purchase, unlocks every other exam and deck.